Convert lbs to kg (÷2.2) first. Calculate mg/kg dose. Compare to safe range. Question anything outside it — every time.
Pediatric Weight-Based Medication Dosing
The calculation that stands between a safe dose and a dangerous one
Unlike most adult medications, pediatric doses are calculated by weight (mg/kg or mcg/kg) rather than given as a flat dose — a critical safety difference nursing students must internalize early. The process: convert the child's weight to kilograms if given in pounds (divide by 2.2), calculate the ordered dose using the mg/kg/dose or mg/kg/day parameter from the order, then compare that calculated dose against the drug's established safe range from a reliable reference. Any calculated dose that falls outside the safe range must be questioned and clarified with the provider before administration — never given as ordered on the assumption the prescriber already checked. Many high-alert pediatric medications also require an independent double-check by a second nurse before administration.
Step 1
Convert weight to kg (lbs ÷ 2.2)
Step 2
Calculate ordered dose using mg/kg parameter
Step 3
Compare calculated dose to established safe range
Step 4
Question/clarify anything outside the safe range
High-alert meds
Require independent double-check by a second nurse
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Flashcard
Weight-Based Dosing
Weight-based dosing — the steps?
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Answer
Convert lbs to kg (÷2.2) first. Calculate mg/kg dose. Compare to safe range. Question anything outside it — every time.
Step 1Convert weight to kg (lbs ÷ 2.2)
Step 2Calculate ordered dose using mg/kg parameter
Step 3Compare calculated dose to established safe range
Step 4Question/clarify anything outside the safe range
High-alert medsRequire independent double-check by a second nurse
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Type 1 Diabetes in Children
3 Ps: Polyuria, Polydipsia, Polyphagia + weight loss. DKA = fruity breath, Kussmaul breathing, abdominal pain. Insulin is never optional, even sick days.
Type 1 Diabetes Mellitus in Pediatrics
The classic presentation — and the rule that surprises new families most
Type 1 diabetes results from autoimmune destruction of pancreatic beta cells, causing an absolute insulin deficiency, and is typically diagnosed in childhood. Classic presentation: the 3 Ps — polyuria (excessive urination, sometimes new bedwetting in a previously dry child), polydipsia (excessive thirst), polyphagia (excessive hunger) — plus unexplained weight loss. If undiagnosed, children can present in diabetic ketoacidosis (DKA): fruity/acetone-smelling breath, Kussmaul respirations (deep, rapid breathing compensating for metabolic acidosis), abdominal pain, dehydration, and altered level of consciousness. Core family teaching: insulin is lifelong and must never be stopped or skipped — including on sick days, when insulin needs often increase due to stress hyperglycemia rather than decrease. Additional teaching: blood glucose monitoring, recognizing hypo- vs. hyperglycemia, carbohydrate counting, and rotating injection sites.
Classic presentation
3 Ps — polyuria, polydipsia, polyphagia — plus weight loss
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Flashcard
Pediatric Vital Signs
Pediatric vitals — and the first sign of shock?
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Answer
Pediatric vitals: HR and RR HIGHER in children, BP LOWER. Tachycardia = first sign of shock in children (BP drops late).
NewbornHR 120–160, RR 30–60
InfantHR 100–160, RR 25–50
ToddlerHR 90–150, RR 20–30
Shock keyTachycardia first → BP drops late
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Developmental Milestones
The 2 · 4 · 6 · 9 · 12 Rule — Smile · Roll · Sit · Pull up · Walk
Developmental Milestones
The motor and language milestones NCLEX uses to screen for developmental delay
Gross motor: 2 months — social smile, holds head up. 4 months — rolls front to back. 6 months — sits with support. 9 months — crawls, pulls to stand. 12 months — walks holding on, may take first steps; walks alone by about 15–18 months. 18 months — runs. 2 years — goes up stairs. Fine motor: 3 months — grasp reflex disappears. 6 months — transfers objects. 9 months — pincer grasp (thumb + forefinger). 12 months — releases objects intentionally. Language: 2 months — coos. 6 months — babbles. 9 months — babbles mama/dada (stranger anxiety, a social milestone, also appears). 12 months — 1–3 words (mama, dada), waves bye-bye. 18 months — 10+ words. 2 years — 2-word phrases. 3 years — 3-word sentences. 4–5 years — sentences, strangers understand speech. Red flags — refer immediately: no babbling by 12 months, no words by 16 months, or any loss of previously acquired skills at any age. Validated tools such as the ASQ-3 are now used for screening (the Denver II is older).
Red flagLoss of any previously acquired skill — refer immediately
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Motor Milestones — Quick Reference
Gross 2 · 4 · 6 · 9 · 12 | Fine 3 · 6 · 9 · 12
Two motor tracks, moving in rough parallel
Which motor milestones are expected at each age?
A fast-recall companion to the Developmental Milestones lesson. Gross and fine motor skills move in rough parallel. The 9-month pincer grasp is the single highest-yield checkpoint.
2 mo
Holds head up
3 mo
Grasp reflex disappears
4 mo
Rolls over
6 mo
Sits with support · transfers objects hand to hand
9 mo
Pulls to stand · pincer grasp (highest-yield)
12 mo
Walks holding on, first steps · releases objects on purpose
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Flashcard
🃏 Motor Milestones — Quick Reference
Gross 2 · 4 · 6 · 9 · 12 | Fine 3 · 6 · 9 · 12
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🃏 Answer
2 moHolds head up
3 moGrasp reflex disappears
4 moRolls over
6 moSits with support · transfers objects hand to hand
9 moPulls to stand · pincer grasp (highest-yield)
12 moWalks holding on, first steps · releases objects on purpose
18 mo / 2 yrRuns / climbs stairs
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Kawasaki Disease
CRASH and Burn — Conjunctivitis, Rash, Adenopathy, Strawberry tongue, Hand/feet swelling, and Burn (fever ≥5 days).
Kawasaki Disease Diagnostic Criteria
Fever ≥5 days plus 4 of 5 CRASH features — and the major exception to "no aspirin in children"
Acute vasculitis, usually under age 5. Diagnosis requires the fever PLUS at least 4 of the 5 CRASH features. Most serious complication: coronary artery aneurysm (20-25% untreated, down to 3-5% with treatment). Treatment: IVIG + HIGH-DOSE ASPIRIN — the deliberate exception to avoiding aspirin in children, since the anti-inflammatory/antiplatelet benefit outweighs Reye's syndrome risk here. Treat within 10 days of fever onset. Delay live vaccines ~11 months after IVIG.
Burn
Fever ≥5 days — the mandatory criterion
CRASH
4 of 5: conjunctivitis, rash, adenopathy, strawberry tongue, hand/feet swelling
Danger
Coronary artery aneurysm — echo required
Aspirin
Deliberate exception to no-aspirin-in-children rule
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Flashcard
Kawasaki Disease
Kawasaki disease — CRASH and Burn
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Answer
CRASH and Burn — Conjunctivitis, Rash, Adenopathy, Strawberry tongue, Hand/feet swelling, and Burn (fever ≥5 days).
BurnFever ≥5 days — the mandatory criterion
CRASH4 of 5: conjunctivitis, rash, adenopathy, strawberry tongue, hand/feet swelling
DangerCoronary artery aneurysm — echo required
AspirinDeliberate exception to no-aspirin-in-children rule
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Cystic Fibrosis
SALT — Sweat chloride test, ADEK vitamins/enzymes, Lung clearance in the right order, Thick secretions cause meconium ileus.
Cystic Fibrosis
"My baby tastes salty when I kiss them" — the classic parent-reported clue
CFTR gene defect → thick, sticky secretions everywhere. Sweat chloride test is the gold-standard diagnostic. Pancreatic enzymes given WITH every meal and snack (not on a fixed schedule) — they only work while food is present. Fat-soluble vitamins A, D, E, K supplemented. Bronchodilator BEFORE chest physiotherapy — opens airways so mucus clears more effectively. Meconium ileus in a newborn (no first stool in 24h) can be an early sign.
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Flashcard
Cystic Fibrosis
SALT
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Answer
SALT — Sweat chloride test, ADEK vitamins/enzymes, Lung clearance in the right order, Thick secretions cause meconium ileus.
SSweat chloride test — gold standard diagnosis
AADEK vitamins + enzymes with every meal/snack
LBronchodilator before chest physiotherapy
TThick secretions → meconium ileus in newborns
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Otitis Media
Pull the pinna — pain gets worse = otitis externa. No change = otitis media.
Acute Otitis Media
A shorter, more horizontal Eustachian tube is why kids get this so often
Diagnosis requires a bulging, immobile tympanic membrane — not redness alone. Pulling the pinna/tragus worsens pain in otitis externa, not true AOM (infection is behind an intact eardrum). High-dose amoxicillin is first-line; switch to amoxicillin-clavulanate if no improvement in 48-72 hours. Watchful waiting (no antibiotics) is an option for nonsevere AOM: unilateral at 6–23 months, any at 2+ years, with follow-up. Pain control is a priority regardless. Tympanostomy tubes for recurrent cases fall out spontaneously in about 6-18 months.
Febrile seizure: common in 6 months–5 years, occurs with rapid temp rise. Simple: <15 min, generalized. Priority: airway, prevent injury.
Febrile Seizures
The most common seizure type in children — NCLEX expects correct priority interventions
Febrile seizures: 6 months–5 years, occur when temperature rises rapidly (fever ≥38.0°C/100.4°F, often higher). Simple febrile seizure: generalized, <15 minutes, resolves spontaneously, no focal deficit. Complex: >15 min, focal, or multiple in 24 hrs. Management DURING seizure: protect from injury (lower to floor, padding), position on side (recovery position), loosen clothing, time the seizure, do NOT put anything in mouth, do NOT restrain. AFTER seizure: assess LOC, check temperature, administer antipyretics, reassure parents. Rectal diazepam (Diastat): if seizure >5 min. Not associated with epilepsy development in most simple cases. Parents need education — very frightening to witness.
Age range
Most common between 6 months and 5 years; fever of 38.0°C (100.4°F) or higher
Simple vs complex
Simple: less than 15 min, generalized, resolves on own. Complex: longer or focal
During seizure
Time it, protect from injury, turn on side, do NOT restrain or put anything in mouth
After seizure
Postictal period is normal — drowsy and confused; monitor and comfort
Parent teaching
Usually benign; small risk of recurrence; antipyretics do NOT prevent recurrence
Infants: car seat, no soft bedding (SIDS). Toddlers: poisoning, drowning, falls. School-age: bike helmets. Teens: MVA, guns, suicide.
Pediatric Safety
Age-specific safety — the leading causes of injury and death at each developmental stage
Infant: SIDS prevention — back to sleep, firm mattress, no loose bedding/pillows/toys, no co-sleeping. Never leave alone on elevated surface. Car seat rear-facing as long as possible, to the seat's rear-facing limit (usually past age 2). Toddler (leading cause of death: unintentional injury): poisoning (lock up meds/cleaners — Poison Control 1-800-222-1222), drowning (never leave alone near water — even bathtub), falls (stair gates, window guards). School-age: bicycle helmets, safety in sports, stranger danger, firearm safety. Adolescent: firearms (#1 cause of teen death since 2020), motor vehicle crashes (#2), alcohol/drugs, suicide. Parents: always know where firearms are stored — lock and store separately from ammunition.
Infants
Rear-facing car seat; no soft bedding (SIDS risk); never leave unattended on surface
Toddlers 1–3
Lock cabinets; pool fencing; outlet covers; remove choking hazards
Preschool 3–5
Supervision near water; harness car seat to its limit (often 65 lb+); stranger safety begins
School age 6–12
Helmet for bike and sports; booster seat until 4 ft 9 in; internet safety; bullying awareness
Adolescents
Seat belt always; driver safety; firearm safety; safe sex education
Kernig signCannot extend knee when hip is flexed — positive = meningeal irritation
Brudzinski signFlexing neck causes involuntary hip and knee flexion
Petechiae/PurpuraRash with meningococcal meningitis = medical emergency
Nursing priorityDroplet precautions until bacterial etiology ruled out; dim lights; quiet environment
TreatmentIV antibiotics ASAP — do NOT delay for LP if patient is unstable
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Asthma in Children
Pediatric asthma: expiratory wheezing, prolonged expiration, accessory muscle use. SABA first (albuterol). Spacer required for children.
Pediatric Asthma
Childhood asthma management — the assessment and stepwise treatment NCLEX expects
Asthma: most common chronic disease in children. Triggered by: URI (most common in children), allergens, exercise, cold air, smoke. Assessment: expiratory wheezing, prolonged expiration, tachypnea, nasal flaring, retractions (intercostal, subcostal, sternal), accessory muscle use, SpO2. Peak expiratory flow: green >80%, yellow 50–80%, red <50% of personal best. Medications: SABA (albuterol/Ventolin): rescue inhaler — use FIRST before exercise or at onset. ICS (inhaled corticosteroid — fluticasone): controller, rinse mouth after (prevents thrush). Children need spacer with MDI. Theophylline: narrow therapeutic index, monitor levels. Status asthmaticus: severe attack not responding to albuterol → IV magnesium sulfate, possible intubation.
Severity signsTripod positioning, inability to speak in full sentences, silent chest = SEVERE
Treatment orderSABA (albuterol) first; moderate-severe: add ipratropium and early systemic steroids
Peak flowLess than 50% personal best = severe; less than 80% = concerning
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Respiratory Distress in Children
Pediatric respiratory distress: nasal flaring, grunting, retractions (subcostal, intercostal, suprasternal), seesaw breathing. Early signs before SpO2 drops.
Signs of Pediatric Respiratory Distress
Recognizing respiratory distress in children — they show signs before oxygen drops
Children compensate well — SpO2 may be normal until they are severely compromised. Assess EARLY signs: Nasal flaring (nostrils widen with each breath), Grunting (physiologic PEEP — keeps alveoli open), Retractions (skin pulls in during inhalation): subcostal (below ribs), intercostal (between ribs), suprasternal (above sternum) — more retractions = more severe. Head bobbing (infants — uses neck muscles), Seesaw breathing (chest caves in, abdomen rises — severe, paradoxical). Stridor: inspiratory = upper airway (croup, epiglottitis). Wheeze: expiratory = lower airway (asthma, bronchiolitis). Always position for comfort — never force a position. Tripod position (leaning forward on hands) = severe distress.
Nasal flaring
Nares widen with inspiration — compensating for increased work of breathing
Grunting
Closing glottis on expiration to create PEEP and maintain alveoli open
Retractions
Subcostal, intercostal, suprasternal — indicate severe work of breathing
Head bobbing
In infants — head bobs with each breath; sign of severe distress
Action
Position of comfort usually sitting up; O2; notify provider; prepare for intervention
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Flashcard
Respiratory Distress in Children
Pediatric respiratory distress — early signs?
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Answer
Pediatric respiratory distress: nasal flaring, grunting, retractions (subcostal, intercostal, suprasternal), seesaw breathing. Early signs before SpO2 drops.
Nasal flaringNares widen with inspiration — compensating for increased work of breathing
GruntingClosing glottis on expiration to create PEEP and maintain alveoli open
RetractionsSubcostal, intercostal, suprasternal — indicate severe work of breathing
Head bobbingIn infants — head bobs with each breath; sign of severe distress
ActionPosition of comfort usually sitting up; O2; notify provider; prepare for intervention
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Pediatric Shock
Tachycardia First — Hypotension Late
HR rises first · BP drops last · Don't wait for hypotension
Children Compensate Longer — Then Crash Fast
Children have excellent compensatory mechanisms — they maintain blood pressure until they've lost 25-30% of their blood volume. By then, they can crash rapidly. The FIRST sign of shock in a child is TACHYCARDIA, not hypotension. Other early signs: prolonged capillary refill above 2 seconds, mottled skin, decreased urine output, irritability. Treat aggressively before hypotension develops.
1st
Tachycardia — earliest and most reliable sign
2nd
Prolonged cap refill, mottled skin, decreased UO
Late
Hypotension — child is in decompensated shock — emergency
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Flashcard
Pediatric Shock
Pediatric shock — which sign comes first, and which is late?
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Answer
Tachycardia First — Hypotension Late
1stTachycardia — earliest and most reliable sign
2ndProlonged cap refill, mottled skin, decreased UO
LateHypotension — child is in decompensated shock — emergency
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SIDS Prevention
Safe Sleep ABCs
Alone · Back · Crib (firm, flat surface)
Back to Sleep — Every Time, Every Nap
SIDS (Sudden Infant Death Syndrome) is the leading cause of death in infants 1-12 months. The AAP Safe Sleep guidelines: Alone (no co-sleeping), Back (supine every sleep), Crib (firm flat surface, no soft bedding, bumpers, or toys). Room-sharing WITHOUT bed-sharing is recommended for at least 6 months. Pacifier use at sleep time is protective. Overheating is a risk factor — dress lightly.
A
Alone — no co-sleeping; room-share without bed-share
B
Back — supine every single sleep, including naps
C
Crib — firm flat surface, no soft items, no bumpers
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Flashcard
SIDS Prevention
Safe Sleep ABCs
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Answer
AAlone — no co-sleeping; room-share without bed-share
BBack — supine every single sleep, including naps
CCrib — firm flat surface, no soft items, no bumpers
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Child Abuse Red Flags
CHILD
Conflicting stories · History doesn't match injury · Injury in non-mobile child · Late presentation · Developmentally impossible injury
Mandatory Reporters — Know These Red Flags
Nurses are mandatory reporters in all 50 states — suspicion alone is enough to report; you do not need proof. Classic red flags: spiral fracture in a non-ambulatory infant (cannot happen from a fall), circular burns (cigarette), bruising on buttocks or back in a non-walking child, delay in seeking care, inconsistent stories between caregivers. Document objectively — exact quotes, exact injury descriptions.
C
Conflicting stories — caregivers' accounts don't match each other
H
History doesn't match — injury severity doesn't fit mechanism
I
Injury in non-mobile child — bruises in infants who can't walk
L
Late presentation — delay in seeking care for serious injury
D
Developmentally impossible — spiral fx in non-ambulatory baby
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Flashcard
Child Abuse Red Flags
CHILD
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Answer
CConflicting stories — caregivers' accounts don't match each other
HHistory doesn't match — injury severity doesn't fit mechanism
IInjury in non-mobile child — bruises in infants who can't walk
LLate presentation — delay in seeking care for serious injury
DDevelopmentally impossible — spiral fx in non-ambulatory baby
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Epiglottitis Emergency
4 Ds — Do NOT touch the throat
Drooling · Dysphagia · Dysphonia · Distress — Position of comfort ONLY
The Pediatric Airway Emergency That Kills Fast
Epiglottitis is a life-threatening airway emergency. The child appears toxic, leans forward in the "tripod position" (sniffing position), drools, cannot swallow, and has a muffled voice. CRITICAL: Do NOT examine the throat, do NOT use a tongue blade, do NOT lay the child down — any stimulation can cause complete airway obstruction. Call anesthesia and ENT immediately. Treat in OR with controlled airway.
!
NEVER examine the throat — can trigger complete obstruction
!
Allow position of comfort — usually tripod/sniffing position
!
Call anesthesia + ENT immediately — controlled airway in OR
SevLethargy, mottling, hypotension — IV bolus immediately
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Congenital Heart Defects
5 Ts of Cyanotic CHD
Tetralogy of Fallot · Transposition · Truncus arteriosus · Total anomalous pulmonary venous return · Tricuspid atresia
Cyanotic vs Acyanotic — The Critical Split
Acyanotic defects (left-to-right shunts) cause extra blood to lungs — heart failure symptoms, not initially blue. VSD is the most common CHD overall. Cyanotic defects (right-to-left shunts) cause deoxygenated blood to bypass lungs — child is blue. Tetralogy of Fallot is the most common cyanotic CHD. "Tet spells" = sudden cyanosis during crying — place child in knee-chest position to increase SVR.
A
Acyanotic — L→R shunt; VSD most common; pulmonary overcirculation
C
Cyanotic — R→L shunt; Tetralogy most common; systemic hypoxia
!
Tet spell: knee-chest position; increases SVR and reduces shunt
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Flashcard
Congenital Heart Defects
5 Ts of Cyanotic CHD
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Answer
AAcyanotic — L→R shunt; VSD most common; pulmonary overcirculation
CCyanotic — R→L shunt; Tetralogy most common; systemic hypoxia
!Tet spell: knee-chest position; increases SVR and reduces shunt
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Pediatric Pain Assessment
FLACC · FACES · Numeric
Under 3 yrs · 3–7 yrs · 7+ yrs
Match the Pain Scale to the Age
FLACC (Face, Legs, Activity, Cry, Consolability) = infants and preverbal children, each scored 0-2, max 10. FACES scale (Wong-Baker) = ages 3-7 who can point but not quantify — 6 faces from smiling to crying. Numeric 0-10 scale = children 7+ who can abstract numbers. Never skip pain assessment because the child is quiet — some children go quiet when in severe pain. Pain is the 5th vital sign.
F
FLACC — birth to 3 yrs; behavioral observation tool
F
FACES — 3 to 7 yrs; point to the face that matches pain
N
Numeric 0-10 — 7 yrs and up; can understand abstract numbers
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Flashcard
Pediatric Pain Assessment
FLACC · FACES · Numeric
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Answer
FFLACC — birth to 3 yrs; behavioral observation tool
FFACES — 3 to 7 yrs; point to the face that matches pain
NNumeric 0-10 — 7 yrs and up; can understand abstract numbers
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Sickle Cell Crisis
HHOP
Hydration · Heat · Oxygen · Pain control
Vaso-Occlusive Crisis — Priority Interventions
Sickle cell vaso-occlusive (pain) crisis is the most common type. Sickling is triggered by: hypoxia, dehydration, cold, infection, stress, high altitude. Priority treatment: IV fluids (hydration prevents sickling), warmth (cold causes vasoconstriction and sickling), oxygen if hypoxic, and aggressive pain management with opioids — do NOT withhold opioids. Aplastic crisis = parvovirus B19 infection + severe anemia.
Children show respiratory distress differently from adults — they use accessory muscles visibly and grunt to maintain PEEP (positive end-expiratory pressure). GRIN: Grunting (expiratory — child is trying to keep alveoli open), Retractions (subcostal, intercostal, suprasternal — all indicate effort), Increased RR, Nasal flaring. A child who has been working hard and suddenly becomes quiet and limp is deteriorating — immediate intervention needed.